Post‑exposure prophylaxis research: mitoquinol and mitochondrial immune signalling in COVID‑19
A pilot clinical trial examining whether Mitoquinol initiated within 72 hours of high‑risk SARS‑CoV‑2 exposure reduces infection rates and symptom duration, including in unvaccinated and immunocompromised individuals.
Recharging the “Immune Battery” in Heart Failure
A double‑blind crossover trial examining whether 4 weeks of Mitoquinol supplementation restores immune cell bioenergetics in patients with heart failure with preserved ejection fraction (HFpEF).
Safety First: Confirming the Absence of Nephrotoxicity at High Doses
A randomised crossover trial assessing whether acute supratherapeutic doses of Mitoquinol produce any evidence of kidney injury in healthy adults, using sensitive renal biomarkers.
Does Mitoquinol Protect DNA from Damage?
A 21‑day supplementation trial examining whether Mitoquinol reduces exercise‑induced nuclear and mitochondrial DNA damage without impairing adaptive training signals.
Acute Functional Rescue: Can Mitoquinol Improve Mobility in Peripheral Artery Disease?
New research explores how an acute dose of MitoQ impacts claudication onset, maximal walking time, and leg artery dilation in patients with Peripheral Artery Disease.
Can MitoQ Rejuvenate Vascular Function in Healthy Older Adults?
A randomised, double-blind crossover trial assessing whether 6 weeks of Mitoquinol supplementation (20 mg/day) improves endothelial function, reduces aortic stiffness, and attenuates mitochondrial oxidative stress in healthy adults aged 60–79.
Boosting HIIT Performance: MitoQ as a Training Amplifier
A double‑blind trial examining whether 3 weeks of Mitoquinol supplementation during high‑intensity interval training (HIIT) enhances mitochondrial and vascular adaptation without blunting exercise‑induced redox signalling.
Restoring Artery Health After Menopause: The “Estrogen Backup”
A controlled study assessing whether a single high dose of Mitoquinol restores flow‑mediated dilation in postmenopausal women and what this reveals about the mitochondrial basis of menopause‑related vascular ageing.

